Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Conversely, our capacity to rescue SERT operate in KI midbrain synaptoneurosomes through the inhibition of FAK implies elevated FAK signaling downstream of your Pro32Pro33 mutant, as verified by elevated pFAK localization in five-HT synapses. https://pro33-login45678.idblogmaker.com/31548961/pro33-login-fundamentals-explained